MEF2 is an in vivo immune-metabolic switch Sir2 acts through Hepatocyte Nuclear Factor 4 to maintain insulin signaling and metabolic homeostasis in DrosophilaĭSir2 deficiency in the fatbody, but not muscles, affects systemic insulin signaling, fat mobilization and starvation survival in fliesĭegradation of arouser by endosomal microautophagy is essential for adaptation to starvation in DrosophilaĪ Drosophila toolkit for HA-tagged proteins unveils a block in autophagy flux in the last instar larval fat bodyĪ somatic piRNA pathway in the Drosophila fat body ensures metabolic homeostasis and normal lifespanĭrosophila cytokine Unpaired 2 regulates physiological homeostasis by remotely controlling insulin secretion
Growth-blocking peptides as nutrition-sensitive signals for insulin secretion and body size regulationĭrosophila lipin interacts with insulin and TOR signaling pathways in the control of growth and lipid metabolism
Steroid hormone signaling is essential for pheromone production and oenocyte survivalįat body regulation of metabolism, organism homeostasis, growth, and developmentĪ nutrient sensor mechanism controls Drosophila growth
Genetic control of the distinction between fat body and gonadalĮnvironmental control of the cell cycle in Drosophila: nutrition activates mitotic and endoreplicative cells by distinct mechanisms Genes involved in tissue and organ developmentįat body and the regulation of metabolismįat body development and maintenance of fat cell survival
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